
 
There's 
no escaping it. Everywhere you turn you can see the evidence, and even if you 
haven't noticed lately, the media is more than happy to remind you, we are a 
nation of lazy super-size me gluttons threatening to undermine the future of our 
health care system with the litany of health problems that will inevitably come 
when the overweight and obese among us start to age.
It's estimated that roughly 
60 percent of Americans are overweight or obese. People are 
finding it more and more difficult to shed those stubborn pounds, no matter how 
hard they try. But the most alarming trend, and one that might offer a clue 
about why the problem is so obstinate, is the rapidly rising rate of childhood 
obesity. For a variety of reasons, kids today are simply not getting the same 
amount of exercise they were getting a generation or more ago. On top of that, 
there's a dearth of choices when it comes to nutritious foods kids can enjoy 
driving them to the nutrient-deficient junk food that typically comprises their 
diet. At first glance it seems that the problem can be easily explained away by 
the combination of these two circumstances. But if you dig beneath the surface 
just a bit, what you will learn might shock you.
Recent studies in the 
area of childhood obesity have revealed some startling findings that have forced 
scientific researchers to look beyond the obvious and long held beliefs about 
weight gain, in order to explain this alarming phenomenon. In 2006 for example, 
scientists at the Harvard School of Public Health reported that 
the prevalence of obesity in infants under 6 months rose a whopping 73 
percent since 1980.
This epidemic of obesity in infants has 
turned conventional wisdom regarding weight gain and loss on its head. Until now 
everyone believed that weight gain was the direct result of consuming 
more calories than you burn -- a perfectly good explanation except when 
applied to babies. Since infants pretty much only consume breast milk and 
formula, the logical conclusion is there must be something else going 
on.
Exposures Add Up
It's no secret that when 
babies are exposed to harmful substances while in the womb it can have 
detrimental effects on their health later in life. This was the case with women 
in the 1950s who were given a drug called DES during pregnancy to prevent 
miscarriage that was later found to increase their daughters' risk of developing 
cervical or vaginal cancer. Scientists are now taking a closer look at what 
substances may be present in pregnant women that could be impacting the later 
development of their children and causing them to be fat.
One explanation 
for the obesity puzzle that's gaining credence is 
early-life exposure to traces of certain chemicals and pollutants in the 
environment --now commonly referred to as endocrine 
disruptors. These chemicals are so similar to human (and animal) 
hormones that they can block or disrupt the actions of regular 
hormones in both.
There is mounting evidence of a strong link 
between early exposure to some of these endocrine disruptors and the later 
development of obesity. Apparently these compounds act on genes in the 
developing fetus and newborn to turn more precursor cells into fat 
cells, which may pre-dispose babies and young children to becoming and staying 
fat. They can also alter metabolic rates, so that the body tends to 
hoard calories rather than burning them. The ability of these endocrine 
disruptors to re-program the fate of a cell and convert it to a fat cell has led 
to the coining of a new phrase to describe them: 
Obesogens.
In one study conducted by Dr. Bruce Blumberg 
of the University of California, Irvine, pregnant mice were fed a substance 
called tributyltin -a disinfectant and fungicide used in marine 
paints and plastics production that enters the food chain through seafood and 
contaminates our water supply. Their offspring were born with more fat 
cells and fat already stored, and became 5 to 20 percent 
fatter by adulthood. The genetic tests revealed that the tributyltin 
activated a receptor, which acts like a switch, determining whether a cell 
remains a fibroblast (cells that make up the body's connective 
tissue) or becomes an adipocyte (a fat cell).
Not all 
endocrine disrupting substances are obesogens, but the biggest culprits so far 
are phthalates, bisphenol-A (also referred to as 
BPA), and some compounds used in stain repellants and non-stick 
cookware. Additional studies in Spain and Belgium have linked fetal exposure to 
pesticides and PCBs with childhood 
obesity.
Phthalates are often found in toys, food 
packaging, hoses, raincoats, shower curtains and other forms of vinyl plastic 
including vinyl flooring, lubricants, adhesives, detergents, nail polish, hair 
spray, shampoo and are often a hidden ingredient in the fragrances found 
in many personal care and body 
products.
Bisphenol-A (or BPA) 
is a hardener that is added to plastic to make it durable and is often found in 
baby bottles, food-storage containers, water coolers, dental fillings, the 
lining inside canned goods, sports equipment, CDs, and even sun glasses, to name 
a few. If it's plastic, doesn't bend and is durable, it's probably BPA. It's 
most often found in polycarbonate (super hard) 
bottles.
Polychlorinated biphenyls 
(PCBs) were used extensively from the late 1940s through the 
late 1970s as coolants and lubricants in electric equipment and have also been 
added to plastics, inks, adhesives, paints, and flame retardant. Although they 
have been banned in most forms and places since 1979, dangerous 
concentrations of PCBs were found as recently as 2010 in soil samples 
from the Great Lakes region, Northeast, Midwest, and Southern United States as 
well as in several European countries.
Looking 
Ahead
What the obesogens research has revealed so far is that 
prenatal exposure to certain chemicals can reprogram an infant's 
metabolism so that it is predisposed to becoming fat. While much of the 
research on this subject is still in its infancy, its implications for the 
future health and well being of our children, and maybe event the adult 
population, are significant and shouldn't be taken lightly.
In light of 
these findings, taking the necessary steps to limit or eliminate your exposure 
to products containing phthalates and BPA seems like a wise thing to do, 
especially if you're pregnant, planning to become pregnant, or have young 
children.
Of course further research will likely be required to determine 
if adult exposure to obesogens has as much of an impact on weight gain. For now, 
most scientists agree that the problem seems to be isolated to fetal exposure 
and exposures during infancy. If you are overweight or an obese adult at the 
moment, it's unlikely that it was the result of early childhood exposures or so 
the thinking goes. Then again, until now, we also believed that the only way you 
gain weight is if you consume more calories than you burn!
Copyright Dropwise Essentials
 Donya Fahmy, is a green business owner and the creator of Dropwise 
Essentials' spa-quality aromatherapy body products that help you safely relieve 
stress, increase vitality, improve confidence, or simply manage your emotional 
state any time or place without popping a pill. For more free tips and valuable 
information visit 
www.dropwise.com and subscribe to the Dropwise Health & 
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